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Cardiovascular

Hearing Loss (SNHL / CHL) and Tinnitus


Differential Diagnoses


Sensorineural Hearing Loss (SNHL)

  • Presbycusis (bilateral, high-frequency loss)

  • Noise-induced (occupational/recreational exposure)

  • Ototoxic drugs (aminoglycosides, loop diuretics, chemotherapy)

  • Sudden SNHL (urgent ENT referral if <72h)

  • Hereditary SNHL (family history)

  • Cerebrovascular ischaemia (AICA involvement)

  • Barotrauma/head trauma (may have CHL component)

  • Ménière’s disease (episodic vertigo, tinnitus, fluctuating hearing loss)

  • Acoustic neuroma (unilateral tinnitus/SNHL)


Conductive Hearing Loss (CHL)

  • Outer Ear: Cerumen impaction, exostosis, SCC (rare)

  • Middle Ear:

    • ETD (URTI, sinusitis, allergies)

    • Otitis media (serous, acute, chronic suppurative)

    • Otosclerosis (stapes fixation, often bilateral)

    • Cholesteatoma (erosive, can be mixed CHL/SNHL)

    • Head trauma (ossicular disruption)

    • TM perforation


Tinnitus

  • Primary (Idiopathic): Typically bilateral, high-pitched, usually SNHL

  • Secondary:

    • Associated with CHL or SNHL (otosclerosis, Ménière’s, cholesteatoma, trauma)

    • Pulsatile tinnitus: Consider vascular lesion (glomus tumour, AV malformation)


When to Image

  • Pulsatile tinnitus

  • Unilateral tinnitus or unilateral hearing loss

  • Suspected middle ear pathology (otosclerosis, cholesteatoma)

  • Significant head trauma


History

  • Onset (acute vs gradual), laterality (uni/bilateral)

  • Noise exposure (occupational, recreational)

  • Family history (early-onset SNHL)

  • Medications (aminoglycosides, diuretics, chemotherapy, salicylates)

  • Trauma (head injury, barotrauma)

  • Associated symptoms:

    • Otalgia, otorrhoea (infection, trauma)

    • Vertigo, imbalance (Ménière’s, labyrinthitis, acoustic neuroma)

    • Aural fullness

    • Facial numbness/weakness (suspect acoustic neuroma, intracranial lesion)


Examination

  • Otoscopy (cerumen, exostosis, perforation, cholesteatoma)

  • Pneumatic otoscopy (assesses TM mobility if effusion suspected)

  • Tuning fork tests:

    • Rinne: AC > BC = normal/SNHL; BC > AC = CHL

    • Weber: Lateralises to affected ear in CHL, better ear in SNHL

  • Cranial nerves (VII, VIII) assessment

  • Audiometry (type, degree, speech discrimination)


Investigations

  • Audiological assessment:

    • Pure tone & speech audiometry

    • Tympanometry (middle ear pressure/compliance)

  • Imaging:

    • CT temporal bone (otosclerosis, cholesteatoma, trauma)

    • MRI IAM (acoustic neuroma, unexplained unilateral SNHL/tinnitus)

  • Bloods (if systemic causes suspected, e.g. autoimmune, syphilis)


Tinnitus Management

  • Address underlying cause (cerumen removal, otitis media treatment, Ménière’s management, medication review)

  • Audiology referral:

    • Hearing aids (if coexistent hearing loss)

    • Sound therapy/masking (white noise, hearing aid noise generators)

  • Psychological support:

    • CBT for distress reduction

  • Lifestyle:

    • Limit caffeine, alcohol, nicotine

    • Avoid loud environments (ear protection if needed)

  • Other interventions:

    • Relaxation, mindfulness, stress management


Notes:

  • Presbycusis & noise-induced SNHL = most common adult SNHL

  • Cerumen impaction = frequent reversible CHL cause

  • Sudden SNHL = ENT emergency (start high-dose corticosteroids if no contraindication)

  • Pulsatile/unilateral tinnitus or SNHL → imaging to exclude vascular/retrocochlear lesion

  • Tinnitus management focuses on education, treating causes, and minimising impact via sound therapy & psychological support

  • Hearing aids can alleviate tinnitus by improving external sound perception

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